近期论文

Nayak G, Cooper GM. (2012). p53 is a major component of the transcriptional and apoptotic program regulated by PI 3-kinase/Akt/GSK3 signaling. Cell Death Dis. 3:e400.
Tullai JW, Tacheva S, Owens LJ, Graham JR, Cooper GM. (2011). AP-1 is a component of the transcriptional network regulated by GSK-3 in quiescent cells. PLoS ONE 6: e20150.
Terragni J, Nayak, G, Banerjee S, Medrano JL, Graham JR, Brennan JF, Sepulveda S, Cooper GM. (2011). The E-Box binding factors Max/Mnt, MITF and USF1 Act coordinately with FoxO to regulate expression of Pro-apoptotic and cell cycle control genes by phosphatidylinositol 3-kinase/Akt/GSK3 signaling. J. Biol. Chem. 286: 36215-36227.
Mullenbrock S, Shah J, Cooper GM. (2011). Global expression analysis identified a preferentially nerve growth factor-induced transcriptional program regulated by sustained mitogen-activated protein kinase/extracellular signal-regulated kinase (ERK) and AP-1 activation during PC12 differentiation. J. Biol. Chem. 286:45131-45145.
Graham, J.R., Hendershott, M.C., Terragni, J. and Cooper, G.M. (2010). mRNA degradation plays a significant role in the program of gene expression regulated by phosphatidylinositol 3-kinase signaling. Mol. Cell. Biol. 30: 5295-5305.
Graham JR, Tullai JR and Cooper GM. (2010). GSK-3 represses growth factor-inducible genes by inhibiting NF-kB in quiescent cells. J. Biol. Chem. 285: 4472-4480.
Terragni J, Graham, JR, Adams KW, Schaffer MW, Tullai JW, Cooper GM. (2008). Phosphatidylinositol 3-kinase signaling in proliferating cells maintains an anti-apoptotic transcriptional program mediated by inhibition of FOXO and non-canonical activation of NFkB transcription factors. BMC Cell Biol. 9, 6.
Tullai JW, Schaffer ME, Mullenbrock S, Sholder G, Kasif S, Cooper GM. (2007). Immediate-early and delayed primary response genes are distinct in function and genomic architecture. J. Biol. Chem. 282, 23981-23995.